Could Aspirin Have Cut COVID Deaths in Half?

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STORY AT-A-GLANCE

  • There was a massive discrediting propaganda campaign hurled at aspirin by Big Pharma fifty years ago when it came out with expensive and dangerous non-steroidal anti-inflammatories (NSAIDs)
  • Aspirin is a staple medicine that is frequently recommended as a remedy to control inflammation and prevent blood clots. It could have helped limit the pandemic death toll, had it not been downplayed and ignored
  • According to research published in April 2021, aspirin reduced COVID-19 patients’ need for mechanical ventilation by 44%, ICU admission by 43% and mortality by 47%
  • Proteolytic enzymes like lumbrokinase, serrapeptase and nattokinase are safer and perhaps even superior choices to aspirin for its anticlot properties. These enzymes, when taken on an empty stomach, act as natural anticoagulants by breaking down fibrin
  • Proteolytic enzymes may also be helpful for long-COVID. Researchers have found that people who die from COVID have extensive lung damage caused by persistent virus-infected cells that cause scar formation. Proteolytic enzymes can help dissolve this scar tissue, as fibrin is a primary component

Aspirin (acetylsalicylic acid) was introduced in 1899 as an alternative to sodium salicylate,1 a pain reliever and anti-inflammatory known for its unpleasant side effects such as stomach cramps, heartburn, nausea and vomiting. It’s been a staple medicine in most households ever since and is frequently recommended as a remedy to control inflammation and prevent blood clots that can lead to stroke and heart attack.

Aspirin also has other health benefits. It helps increase the oxidation of glucose as fuel for your body while inhibiting the release of fatty acids from your fat cells, specifically linoleic acid (LA), an omega-6 fat which I suspect is a primary driver of chronic disease.

This is important because nearly everyone in the U.S. has excessive LA in their tissues, as it takes seven years of a low LA diet to get it down to healthy levels. So, the last thing you want to do is increase the release of LA into your body from fat stores. It is far better to release LA slowly and allow your liver to process it. It is water soluble, so you can urinate it out without it being metabolized into inflammatory prostaglandins.

Importantly, aspirin will also lower your baseline cortisol — indirectly by lowering inflammation, and directly by inhibiting the enzyme 11-beta-hydroxysteroid dehydrogenase Type 1. This enzyme synthesizes active cortisol from the inactive precursor cortisone.

Aspirin lowers the production of stressed induced aldosterone, which can help to lower blood pressure. Aspirin increases your levels of carbon dioxide and progesterone while inhibiting the major inflammatory pathway, NF kappa-B, which will help your body naturally increase the synthesis of two powerfully important hormones that your body needs, testosterone and progesterone.

 

Aspirin also uncouples mitochondria. Uncoupling of mitochondrial oxidative metabolism from ATP production can help to increase your metabolic rate and help you lose weight. Dinitrophenol (DNP) is a drug that, like aspirin, uncouples mitochondrial metabolism and produces incredible weight loss. Sadly, it has a very low therapeutic index, so its effective dose is close to its toxic dose and is widely considered too dangerous for clinical use and is no longer available in the U.S.

Aspirin Reduced COVID-Related Hospital Deaths by 47%

Aspirin could also have helped limit the pandemic death toll, had it not been downplayed and overlooked. Many news outlets and COVID-specific websites warned against…

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